Oncolytic measles virus expressing the sodium iodide symporter to treat drug-resistant ovarian cancer.
By: Evanthia Galanis, Pamela J Atherton, Matthew J Maurer, Keith L Knutson, Sean C Dowdy, William A Cliby, Paul Halulska, Harry J Long, Ann Oberg, Ileana Aderca, Matthew S Block, Jamie Bakkum-Gamez, Mark J Federspiel, Stephen J Russell, Kimberly R Kalli, Gary Keeney, Kah Whye Peng, Lynn C Hartmann

Division of Medical Oncology, Mayo Clinic galanis.evanthia@mayo.edu.
2014-11-16; doi: 10.1158/0008-5472.CAN-14-2533
Abstract

Edmonston vaccine strains of measles virus (MV) have significant antitumor activity in mouse xenograft models of ovarian cancer (OvCa). MV engineered to express the sodium iodide symporter gene (MV-NIS) facilitates localization of viral gene expression and offers a tool for tumor radiovirotherapy. Here we report results from a clinical evaluation of MV-NIS delivery in patients with taxol and platinum resistant OvCa. MV-NIS was given intraperitoneally every 4 wk for up to 6 cycles. Treatment was well tolerated and associated with promising median overall survival in these heavily pretreated OvCa patients: no dose limiting toxicity was observed in 16 patients treated at high dose levels (108 -109 TCID50). Median overall survival of 26.5 months compared favorably with other contemporary series. MV receptor CD46 and nectin-4 expression was confirmed by IHC in patient tumors. Sodium iodide symporter expression in patient tumors after treatment was confirmed in 3 patients by 123I uptake on SPECT/CTs and was associated with long progression-free survival. Immune monitoring post-treatment showed increased levels of T cells recognizing the tumor antigens IGFBP2 and FRα, suggesting that MV-NIS treatment triggered cellular immunity against the patients' tumor and supporting an immune mechanism in mediating antitumor effects. Our findings support further clinical evaluation of MV-NIS as an effective viral immunotherapy.



Copyright © 2014, American Association for Cancer Research.

PMID:25398436






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